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Looking for someone else? One of the biggest complaints I hear from women is that their partner, boyfriend, husband, whatever bought them the biggest vibrator or dildo that they had ever seen. And while the common thought is that bigger is better, this isn’t always the case for every woman. Some women aren’t designed for that kind of load capacity, so you’re going to need to think a little more compact, perhaps. Shoving something big into something that is small isn’t pleasurable in the least bit. And the sad part is that a lot of the women won’t even say anything because they don’t want to offend your choice. Why make these poor ladies suffer? Be sensible about size unless you know she can take it! For some os us, bigger is indeed better... Look at yourself. Yes, unzip your pants and look at what your woman is already accustomed to feeling inside her. Be honest, this isn’t the time to start bragging about the size, length, whatever, no one is looking but you. That’s the size that you want to choose for a vibrator for your woman. Slightly bigger is okay too, but don’t go for the foot in diameter version. Her eyes may widen when she sees it, but that’s in fear, not anticipation. Looking for yourself? A penis pump can be a great way to stimulate yourself when you’re flying solo for a while, but you have to give this purchase a little thought as well. A lot of these pumps are not designed for all sizes of men. You want to read the package carefully to see what the design is. Sometimes there is a cup at the end of the penis pump that will catch your juices as well as stimulate the tip of the penis. Be a little wary of the texture that a pump has - it can be irritating if the material sticks to places where it just shouldn’t stick. So use LOTS of lube! Does that still seem stimulating? The lashings of lube sensation can be very different if you haven’t tried it… If you’re here online, you’ll want to read the reviews on the site. These folks aren’t paid to give their opinions, so you’re sure to read the truth. And if you’re still not sure about the product, ask! The touch, the feel of vibrators When it comes to vibrators, the material can make all of the difference. It can be smooth or textured in the same way as a real penis. There are also others that come with special textures and grooves for her ultimate pleasure. These are supposed to hit the g-spot and though each woman is different, they can help in other ways. If you end up buying a vibrator, a little lube can help here as well. The extras that matter Oh, and I almost forgot— why not find a vibrator that’s waterproof and has a lot of speed settings. Waterproof comes in handy because it can be used in pools and bathtubs. And who wouldn’t like a little pick me up in the morning shower? Or for the ultimate private experience, use the (waterproof models only!) vibrator in the tub—it’s ultra quiet and discreet. Speed settings help to control the pleasure of the experience, plus the vibrator can then be used on the clitoris as well. With a little lube and a good vibrator, the woman can feel waves of orgasms—repeated over and over. Yes, multiple orgasms are possible—especially with a good vibrator. And when the man has picked out a great vibrator for himself or for his partner, they’re going to be thanked in very special ways. For more information about lingerie please have a look at this link:Exotic Lingerie | Blouses See Thru free penis enlargment pills vimax truth about penis enlargement pills vimax best penis enlargement enlargement penis pill vimax do penis enlargment pills work penis elargement excersizes cheapest penis enlargement pills penis enargement program

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Viagra (Sildenafil citrate), which millions of men take for erectile dysfunction (ED), reduces the effects of hormonal stress on the heart by half, according to a study published online in the journal Circulation. Viagra causes genital blood vessels to expand, which helps in maintaining an erection. Recent research also has pointed to its potential usefulness in treating pulmonary hypertension. Prior to the latest findings by a team of Johns Hopkins researchers, it was thought to have little effect on the heart. Viagra, or sildenafil, blunts the strengthened heart beat caused by chemically induced stress, according to study senior author and cardiologist David Kass, MD, a professor at the Johns Hopkins University School of Medicine and its Heart Institute. It thereby lessens both the excess amount of blood and the force used to pump it to the body. "Sildenafil effectively puts a 'brake' on chemical stimulation of the heart," says Kass. Prevents and Reverses Effects of High BP These findings are believed to be the first confirmation in humans that Viagra has a direct effect on the heart. In earlier research, Kass and his team observed a similar effect in mice; Sildenafil blocked the short-term effects of hormonal stress in the heart. Related studies by the group show that sildenafil also prevents and reverses the long-term effects of chronic high blood pressure on the heart. Sildenafil reversed the negative effects on heart muscle weakened by heart failure and enlargement -- a condition called hypertrophy -- in mouse experiments Kass and his team carried out earlier this year. They reported their results in the journal Nature Medicine. "But we had no firm evidence as to whether or how this therapy might work in the human heart," says Kass. "Our latest research provides firm evidence this drug does indeed have an important impact on the heart." Increased Heartbeat Was Slowed Thirty-five healthy men and women, with an average age of 30 and no previous signs of coronary artery disease, participated in the six-month Johns Hopkins study. Within a three-hour timeframe, each participant received two separate injections of dobutamine (5 micrograms per kilogram for five minutes), a synthetic, adrenaline-like chemical that increases heart rate and pumping strength. Between injections, study participants were assigned randomly to a group that was treated with sildenafil (100 milligrams taken orally) or to a group given a sugar pill placebo. All participants then were given the second dobutamine injection to see what effects sildenafil or placebo had on the heart. Measurements of heart function were made before and after each injection. These included blood pressure readings, electrocardiograms and echocardiograms. Blood samples confirmed relatively equal levels of sildenafil and other enzymes. Each dobutamine injection stimulated heart function, increasing heart rate and the force of each heartbeat used to pump blood throughout the body, results showed. "This stimulation is similar to the way the nervous system normally increases heart function when triggered by emotional or exercise stress, or in diseases such as heart failure," notes Kass. After the first injection of dobutamine, the force of heart contraction increased by 150 percent in both groups. In the placebo group, this increase repeated itself after the second injection. However, in the group treated with sildenafil, the increased heartbeat was slowed by 50 percent, resulting in a smaller increase in blood flow and blood pressure generated by the heart in response to chemical stimulation. Between injections, heart function was not altered in the sildenafil group, demonstrating the absence of adverse side effects on the resting human heart. Stops PDE5A Action "Knowing more about the effects of sildenafil on heart function will allow for safer evaluation of its use as a treatment for heart problems," says Kass. "Our results set the stage for further studies of sildenafil's immediate and long-term effects on the heart and its ability to modify other neurohormonal and stress stimuli, including adrenaline and hypertension," he adds. While the precise biological actions of sildenafil in the heart are not fully understood, the drug is known to work by stopping the action of an enzyme, called phosphodiesterase 5 (PDE5A), Kass explains. This enzyme is involved in the breakdown of a key molecule, cyclic GMP, which helps control stresses and limit overgrowth in the heart. PDE5A is also the biological pathway that sildenafil blocks in the penis to prevent the relaxation of blood vessels and thus maintain erections. Copyright 2005 Daily News Central penile enlargement surgeries penis enlagement supplement easy enlargement free penile surgery way penile enlargement before and after photo pnis enlargement information real penis enhancement free natural penis elargement penis enlargment surgeon penis elargement surgery cost

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. do penis enlargement pills work vimax penis enlargement pump best enlargement exercise penis penis enlarement technique penile enlargment surgeries natural penis enargement pills natural penis enlargement pills pnis enlargement drug penis elargement surgery cost

The Stone-Builders [By their Weapons] [Big-chest was a reluctant hero, for the most part, that is, in killing the Stone-Builders; yet when he could, he did so of course, --but I say reluctant I suppose with reluctance; for it was not unlike everyone at this time to be disinclined to kill them, they seemed to be in the vein of the gods, un-killable: yet he killed them and fought them, more so than anyone else—less out of conviction than out of desire to avoid dishonor and social embarrassment for himself.] Said I [I, being: Short-legs], I had seen one time Big-chest walk into a campsite of theirs, the Stone-Builders that is, —I tried to tell this story to my brother, Stern-toes, once, but I never could explain it right, but I think he got the jest of it, if not the seriousness, we did both laugh at the Stone-Builders for hours on end, afterwards. As I was about to say, myself and Little-eyes witnessed this whole happening from a distance of course. The Stone-Builders were full of what they called: ‘wone, or wine,’ something along that order, some sounds take me back a bit, they had new sounds all the time, yes O yes, inventing new sounds like the growing of leafs on a tree, coming into our brains like new winds, dragging it into a mode of thinking more everyday, instead of being fond of the birds, and just living: eating, sleeping and dying—these words we never heard of before were floating everywhere in the air, ever since that is, the arrival of the Stone-Builders on the scene: before this, before Eve walked out of that Garden, things were dangerous, but much more quiet. Well, Big-chest, noticed one evening—not being too far in the thick of the foliage by their campsite [the Stone-Builders site]—they had killed a man-eater [lion]; there were four of them, called ‘soldiers,’ at the campfire-site, a resting place to them: just laughing, and drinking, and being playful like a group of little cubs: lion cubs—wild and whimpering [whiny] lion-cubs, that is exactly what they were like. I could see Big-chest laughing to himself—inside that big muscular oversized chest: as he watched them wrestle around with one another, actually they did get a little over physical with one another, like the wild boars whom would chase after one of us, wanting to eat us for a feast, and then they’d settle down again. It was a cold winter’s night that day, so there was a real chill in the air, and not all that much leafy undergrowth for us to hide or for that matter to slap the chill away: nor for that matter Big-chest: although he had a coat of hair all over him to keep him warm anyways—showed a bit of chill in his face also. Although—I was grateful for the few large trees with the plant-life tucked around me, it absorbed some of the wind—the brisk, cold winds seeping by us, around us, and almost through us: the shrubbery covered us, as we remained in the distant woods, with a pile of leaves up to our knees: leaves for warmth and camouflage, --camouflage being a plan incase we figured we’d have to duck, hide ourselves-quickly should they get the best of Big-chest, but we doubted that: Big-chest was just the opposite of us----mean,--plus as always, Big-chest was confident of his abilities, he stood in the woods, no shadow—not sure what he did with it, but he was cleaver; just a big blob of muscle, hair and sharp eyes, small squinty eyes pinned on the four Stone-Builders, at the camp site, and their man-eater, that was going to be his dinner. He was actually blocking our vision a bit, but I think he did that because he wanted to show his audience—which was us—who was the braver. I actually had some kind of a feeling for those men who were about the meet Big-chest, a gloomy feeling at best, and a thankful feeling: thankful, it was not us in their place; yet both I and Little-eyes, both surely held feelings of revenge for the Stone-Builders, and this was kind of a good time for the revenge to seep out, but I don’t care for revenge for the most part, not really, it takes too much energy, saps you—in review, all was quite mysterious to me. Then unsuspected, he walked into the camp, tall as a mountain, hairy as a leafy think forest, long, a very long mouth from ear to ear, his teeth showed—he walked reminiscent of the king of the Stone-Builders [I think he was mimicking him—he like to do such things], he must have seen him walk, for he was arched just like their king, head back, eyes slanting down as if they were subordinates; --among the four he crept up, not a word, not a sound, the dark-dragging behind him, the sky had very little light given by the stars, but it followed him overhead none the less, a cloud covered the moon—as if he and Big-chest were pals; now he had seen their weapons by the fire, where the dead lion lay, if anything, Big-chest was shifty: sly, observant: he was swaying his body akin to the huge trees in a storm, not sure exactly why, but I think it got his blood moving and his limbs more flexible for swinging when he used them for clubs—and it made for a good showing: his hands were as big as large branches of a tree: and as hard. Closer and closer he came to the fire, no one noticed him yet, can’t figure it out, no one, no one at all, --could they not hear him a little, just a tiny bit, I asked myself, for both myself and Little-eyes could hear his foot steps even in the woods, at twice their distance, I was about to learn we had better hearing than these new creatures. But then this new breed of course, can not have all the advantages, thank goodness, thus, our senses were better, we were tuned higher one might say, and they were tuned with more and a higher intelligence than we. His fingers now, almost touching the ground—I could see—he, he had long thick arms, and fingers, and perturbing muscles, he was impressive to look at, huge to digest with your eyes, and frightening if you did not see him on a daily bases, and dangerous to be around, at any time. Then all of a sudden two of the four turned their necks to see what was in back of them; not sure if they heard him, sensed him, or just did out of an automatic military checking ritual,--whereupon, they almost went into shock: two stood up, all four were some fifteen feet from their weapons. The two who were squatting, the closest to the fire, were in a panic, the other two were a little farther away, standing now, unsure, thinking. I think one was releasing himself; he made a puddle and was trying to cover it up by kicking dirt, how modest. I figured why waste your time, this was precious time, run, run, run: that is what I’d do, but I really was hoping they’d not run, I must have an evil side in me also, just like them; you know, they got this pride thing, and I was hoping they would stay with this pride and arrogance, and then as I stopped thinking for a moment, Big-chest knock it out of them, if that is, they had any pride left. I think I was starting to get like them, that being: aggressive thoughts. In any case, Big-chest took his right hand swung it backwards to build up momentum, and with the force of a giant tree, hit the head of one of the squatters as he was about to stand up, it sounded faintly similar to thunder, and I could hear it snap, and rip, similar to a timber falling after lightening strikes it, strikes a tree out of its roots, its stretching roots out of the ground. He fell on his chest, then pushing himself, flopped over and onto his shoulder as if it had nothing holding his head in place, like a dead fish flopping, jumping in a creek—he lost his inner breath. The other one tried to get to his weapon, but Big-chest, akin to lightening, jumped with one leap over to him, picked him up by one leg, his penis showing, as Big-chest looked strangely at it, as if to laugh at a small ugly worm, for they all liked covering them up for some odd reason, and Big-chest now must have figured out, he knew why. And we both in the bushes started to giggle, snicker, laughing at the sight—I wanted to say laugh again—but we had to hold our laughing inside our stomachs for a while, so as not to spoil his feat. Then after our expressions of amusement, a stern grin appeared on Big-chest’s face—I think he heard us—in any case, he tossed him into the fire when he got bored looking at him, after twisting him about for a few seconds, breaking his leg in several places I imagine, for I kept hearing crunches, as if bones were cracking, and then there was his screams. Then one of the two standing routed himself through the woods yelling something on the order of: “Hhhhh eel pppp...!” Not sure what that meant. The last one, I call him the brave one, or definitely I could call him the stupid one, or should I say foolish one, none-the-less, he pulled out a sharp object, about the length of his hand, and stood in front of Big-chest as if he was going to fight him. At this point I said, and Little-eyes thought: this was the end for him; he [the soldier] looked like a banana compared to Big-chest. I asked myself, ‘Is he crazy? Run, and run while you can,’ and I was on Big-chest’s side now, more than ever, but it didn’t sound like it for that split-moment, but I felt it was a little unfair, size and all. But the man, whom I am calling a brave-soldier, stood his ground, and actually looked at Big-chest in the eyes. My-gosh, the man must have been half his size, about 175 pounds, quick on his feet though, for he was dancing around Big-chest, trying to stab him, and poke him. He looked more like a bee trying to sting someone, but that just irritated him more. Big-chest had taken arrows out of himself one-hundred times before, I bet; arrows deeper than that knife would have ever penetrate, if the person had gotten a chance to lunge it into Big-chest, and he didn’t get that chance: and it never hurt him much: those pokes. These little wounds were nothing, --but should he leap and get a good stab possible in the upper chest of Big-chest, or eye, then I’d worry. To make a long story short: Big-chest just looked dumfounded at the figure in front of him dancing in a circle, and didn’t move very much, except around; I’ve seen Little-eyes close his eye-lids now, he knew, he knew what was about to happen, and with his waving quick long arms, Big-chest picked up the seven foot lionesses, and put it over his shoulder, the crazy Stone-Builder charged at him, and Big-chest with a quick sweep, with a turn, knocked the man flat on his back, onto the ground, he had hit him with the man-eater, as he balanced it over his shoulder. Then, somewhat, disparate, or so it seemed, reminiscent of a dying fish jumping about trying to get back into the water—he: Big-chest—kicked him in the mid-section of his belly, sweeping him into the fire like trash, now almost a dead fish. The Soldier could not move, he surely had a broken spine I thought, had he not, he would had gotten up and run fast out of the fire, and he didn’t: or couldn’t, for Big-chest couldn’t run with the man-eater on his shoulder so it was a good time to escape, if he could. But he didn’t, or couldn’t, nor do I think he intended to. But again, the man tried to move out of the fire with no suitable means other than his arms which were now on fire, for surely his ribs and legs were broken. Big-chest simply turned away from him as if he was insignificant, as I did myself. The defeat was predictable, and most unnecessary. I got thinking: what kind of creature fights when they cannot compete. It has always been the law of the land—to run, unless cornered: hence, when you can’t battle, don’t. It wasn’t necessary to die like that. I was learning about pride and arrogance quickly from these new creatures though; all in time and observation I told myself, and I’d be well informed on their unusual habits. 11 Early winter We had no way of knowing which winter would be good to us or bad for us, and winter this particular winter had come early, and therefore our food supply was exhausted, depleted that is, rather quickly. When Little-eyes and I returned back to the cave the following evening, we had told in our symbolic way: expressed at the Banana Cave that is, to the entire Horde how Big-chest had killed the Eve People. And you could hear the laughter for miles around. I tried to explain how Big-chest had seen or sensed their movements, their evil objective, and their killing intent: as he always seemed to be able to sense survival quite well; he had a special quality of seeing through a person to his evil side, as he could see through us, thus, he could see through the Stone-People as well. I explained how one of the men stayed to fight him, trying to outstare Big-chest, and got kicked into the fire, and died. They all shook their heads in wonderment, we were not the smartest of the inhabitants of earth, but that was sure dumb we all thought, no vocal language was needed for that understanding or response. I think Big-chest had taken his trophy to his cave in our area, and was having a formal meal at this time. We liked anyone who could out smart the Stone-People I suppose, they were smug and we were helpless to them most of the time; they had well groomed weapons, and we had simply rocks and some clubs, along with a few sharpened stones, as they now were being called, knifes, up to the appearance of the Stone-People, they were just tools. And so it felt good if anything, good to see the odds turn for once, and to be frank, they didn’t turn much, if ever in our favor after that episode. But our surprise would come in the morning: --yes, we would not be forgotten for once. Morning In the morning when several of us looked out of our cave entrances, in the center of the canyon below our cliff dwellings, as we often did to be sure we were safe from man or beast, in the open area in the valley below us, we saw half a lion torn open, lying in the center of our domain, for us, it was a treasure, a gift, a donation if anything, and all of us quickly ran to eat what meat Big-chest had left for us. Big-chest was not always so generous, or kind, but for some odd reason, he knew we were starving for some protean, and our bodies were starting to show our ribs. Aimless to say, this never happened again—not in such a quantity, but we all gave Big-chest a super big smile as we walked proudly out of our canyon-caves and ate the raw meat [for he appeared standing erect by a cave entrance observing the feast he provide]; yes, some of us even were tarring at the red meat, animal protein, liken to wolfs. 12 The Hermit by the Sea It was a short period of time from when Big-chest appropriated the lion [took it from the Stone-Builders] and we all ate the meat, when I joined the Horde in the valley on a crisp morning—a morning that told me, the seasons were about to change, thus, leading into spring; I could see my breath: it was so brittle, so I knew winters end was near. There was great commotion in the valley below, as there often was when someone or something new came about to celebrate, I had noticed from my cliff dwelling a gathering of the Horde, looking down, I quickly dashed along the sides of the cliff until I reached the floor of the valley to see what it was, as did Little-eyes, as I had woke him, trying to explain a happening was taking place. Thin-hips of the Horde [Sister to Moss] When I reached the bottom and many of the folk were going to and fro, some with sad and hungry faces, very sad posture, I made my way through several folks now gathered around this one section of the cliff; old-Moss, the Hermit by the Sea, was laying dead, his sister, Thin-hips, was there pacing, walking back and forth, kind of chanting, humming something, sounds on top of sounds—death had waxed his face I noticed. Old Moss was the oldest folk I had ever known, ever heard of. He must have been 60 or 65 years old—I doubt Big-chest was that old. No one ever lived that long, no one that is but Moss, I suppose. You could tell by looking at him, half his death was caused by starvation, the other by his long walk back to the Valley of the Caves, the strenuous walk; a walk many took to come back when they felt their time was short on this ground, like some fish, we all seem to know our dying ground; he came from the far off place, called the Great Cliffs by the Sea. I had only seen him when I was a kid and then once or twice coming and going, within a twenty-year period. He lived in the sand hills far from the Horde as I was saying, to the extreme East, and not far from there to the south was the Great Sea and the cliffs he always told his sister about, much larger than ours, higher than ours he’d say. He add, this place was somewhere between the Sea, and the cliffs, and the strait, and this valley was a flat area, plateau, this is where he wondered off too often, or so he’d claim, upon his return. He knew my father quite well, Long-arms, and did visit him, it was always when I was gone it seemed. They appeared to get along quite well, as one might expect, two strange folks to say the least; not sure what they had in common, matter of fact, if anything, one was lazy—my father, the other, Moss, was quite active I heard. The Great Sea But he did bring back information to his sister, who shared it with us, and of course he’d tell other people also of his journeys, or try to describe them best he could, and we were all quite interested in his tales—it was entertainment: yes he was a man of tales, I guess in one way I admired him for that, it was almost like some of the occupations the Stone-Builders had, or called occupations, which were really doing things by order of their king and getting fed by someone else because of the king—strange. Thus, Moss was our entertainer, and Moss did get fed by most of the Horde’s residents for doing so, I think they’d call him in to their cave to hear him talk, or draw pictures, or act out his strange adventures. Half the time we never knew what he was saying, but then, so what and it was amusement. Everyone liked him, and so did I. penis enlagement pills best penis elargement surgery penis enhancement pic before and after manual pennis enlargement penis girth enlargement penis enhancement forum vimax penis enlargement tool penis enlarement surgery penis elargement surgery cost

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